A rare genetic condition affecting the function of cilia in various organs of the body
Cilia are microscopic, hair-like structures that line the airways (including the nose, sinuses, and lungs), middle ear, and reproductive tract. In healthy individuals, cilia beat in a coordinated manner to clear mucus and trapped particles from the airways, helping to prevent infection and maintain respiratory health.
PCD affects approximately 1 in 15,000-30,000 individuals, making it a rare genetic disorder.
PCD is caused by mutations in genes responsible for the structure and function of cilia. Currently, more than 40 genes have been identified that can cause PCD when mutated. The condition is inherited in an autosomal recessive pattern, meaning an individual must inherit affected copies of the gene from both parents to develop PCD.
Common PCD genes include DNAH5, DNAI1, CCDC39, and CCDC40, which affect different parts of the ciliary structure.
PCD affects multiple organ systems due to the widespread presence of cilia throughout the body. Common symptoms include:
The combination of situs inversus, chronic sinusitis, and bronchiectasis is known as Kartagener syndrome, a subtype of PCD.
Diagnosis of PCD typically involves multiple tests, as no single test can definitively diagnose all cases. Common diagnostic approaches include:
Early diagnosis is important for implementing treatments that can help prevent long-term complications like bronchiectasis.
While there is currently no cure for PCD, treatment focuses on preventing and managing symptoms through:
With proper management, most people with PCD can lead full and active lives, though regular medical care is essential.